Improvement of tardive dyskinesia following amisulpride treatment

dc.contributor.authorPeritogiannis, V.en
dc.contributor.authorTsouli, S.en
dc.contributor.authorZafiris, S.en
dc.contributor.authorPappas, D.en
dc.contributor.authorMavreas, V.en
dc.date.accessioned2015-11-24T19:11:22Z
dc.date.available2015-11-24T19:11:22Z
dc.identifier.issn0278-5846-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/20923
dc.rightsDefault Licence-
dc.subjectAntipsychotic Agents/*adverse effectsen
dc.subjectDyskinesias/*etiologyen
dc.subjectFemaleen
dc.subjectHumansen
dc.subjectMiddle Ageden
dc.subjectSchizophrenia, Paranoid/drug therapyen
dc.subjectSulpiride/adverse effects/*analogs & derivativesen
dc.titleImprovement of tardive dyskinesia following amisulpride treatmenten
heal.abstractTardive dyskinesia (TD) is a persistent, disturbing side effect of antipsychotic drug treatment. It is established that clozapine and other second generation agents cause less TD and may also improve pre-existing TD. We report a case of significant improvement of TD after the administration of amisulpride, a potential D(2)/D(3) antagonist that has atypical properties. The possible effect of fast dissociation of D(2) receptors is being discussed.en
heal.accesscampus-
heal.fullTextAvailabilityTRUE-
heal.identifier.primary10.1016/j.pnpbp.2006.01.022-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/16574293-
heal.identifier.secondaryhttp://ac.els-cdn.com/S0278584606000480/1-s2.0-S0278584606000480-main.pdf?_tid=39059967b113da99da4f273f61605fe8&acdnat=1332941369_64fedc87917f57539cdbd11855ecc3e6-
heal.journalNameProg Neuropsychopharmacol Biol Psychiatryen
heal.journalTypepeer-reviewed-
heal.languageen-
heal.publicationDate2006-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.typejournalArticle-
heal.type.elΆρθρο Περιοδικούel
heal.type.enJournal articleen

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