Esophageal dysfunction in chronic hemodialysis patients

dc.contributor.authorSiamopoulos, K. C.en
dc.contributor.authorTsianos, E. V.en
dc.contributor.authorDardamanis, M.en
dc.contributor.authorBerecos, C.en
dc.date.accessioned2015-11-24T19:18:25Z
dc.date.available2015-11-24T19:18:25Z
dc.identifier.issn0028-2766-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/21864
dc.rightsDefault Licence-
dc.subjectAdulten
dc.subjectEsophagus/physiology/*physiopathologyen
dc.subjectFemaleen
dc.subject*Gastrointestinal Motilityen
dc.subjectHumansen
dc.subjectKidney Failure, Chronic/*physiopathology/therapyen
dc.subjectMaleen
dc.subjectManometryen
dc.subjectMiddle Ageden
dc.subjectMuscle, Smooth/physiology/physiopathologyen
dc.subject*Peristalsisen
dc.subjectReference Valuesen
dc.subject*Renal Dialysisen
dc.titleEsophageal dysfunction in chronic hemodialysis patientsen
heal.abstractEsophageal motility was studied in 16 asymptomatic hemodialysis patients and 38 normal volunteers. The incidence of triphasic tertiary and biphasic contractions was significantly higher in the patients than in the control subjects (25 vs. 0% and 87.5 vs. 39.5%, respectively). The above abnormalities were recorded in the lower two thirds of the esophageal body. We suggest that manometric evidence of esophageal dysfunction is very common in such patients. Although the pathogenesis is unclear, uremic neuropathy of the vagus nerve and/or smooth muscle myopathy due to chronic uremia could be the causative factors.en
heal.accesscampus-
heal.fullTextAvailabilityTRUE-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/2392192-
heal.identifier.secondaryhttp://content.karger.com/ProdukteDB/produkte.asp?doi=10.1159/000186006-
heal.journalNameNephronen
heal.journalTypepeer-reviewed-
heal.languageen-
heal.publicationDate1990-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.typejournalArticle-
heal.type.elΆρθρο Περιοδικούel
heal.type.enJournal articleen

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