Oesophageal dysfunction in patients with primary Sjogren's syndrome

dc.contributor.authorTsianos, E. B.en
dc.contributor.authorChiras, C. D.en
dc.contributor.authorDrosos, A. A.en
dc.contributor.authorMoutsopoulos, H. M.en
dc.date.accessioned2015-11-24T19:01:13Z
dc.date.available2015-11-24T19:01:13Z
dc.identifier.issn0003-4967-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/19658
dc.rightsDefault Licence-
dc.subjectAdulten
dc.subjectAgeden
dc.subjectEsophagogastric Junction/physiopathologyen
dc.subjectEsophagus/*physiopathologyen
dc.subjectFemaleen
dc.subjectHumansen
dc.subjectManometryen
dc.subjectMiddle Ageden
dc.subjectPeristalsisen
dc.subjectSjogren's Syndrome/*physiopathologyen
dc.titleOesophageal dysfunction in patients with primary Sjogren's syndromeen
heal.abstractOesophageal motility was studied in 22 patients with primary Sjogren's syndrome and 20 normal volunteers. Oesophageal dysfunction was detected in eight of the 22 patients (36.4%) with primary Sjogren's syndrome. No abnormalities were detected in the normal subjects. Individual analysis of the oesophageal motility studies showed different patterns of oesophageal dysfunction; aperistalsis (three patients), triphasic tertiary contractions (two patients), frequent non-peristaltic contractions (two patients), and low contractions (one patient). These oesophageal abnormalities did not correlate with the parotid flow rate, the degree of inflammatory infiltrate of the minor salivary glands, the extraglandular manifestations, or the presence of autoantibodies.en
heal.accesscampus-
heal.fullTextAvailabilityTRUE-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/4037887-
heal.identifier.secondaryhttp://ard.bmj.com/content/44/9/610.full.pdf-
heal.journalNameAnn Rheum Disen
heal.journalTypepeer-reviewed-
heal.languageen-
heal.publicationDate1985-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.typejournalArticle-
heal.type.elΆρθρο Περιοδικούel
heal.type.enJournal articleen

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