Hypouricaemia as a marker of a generalized proximal tubular damage in alcoholic patients

dc.contributor.authorLiberopoulos, E.en
dc.contributor.authorMiltiadous, G.en
dc.contributor.authorElisaf, M. S.en
dc.date.accessioned2015-11-24T19:12:31Z
dc.date.available2015-11-24T19:12:31Z
dc.identifier.issn0735-0414-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/21078
dc.rightsDefault Licence-
dc.subjectAgeden
dc.subjectAlcoholism/*blooden
dc.subjectHumansen
dc.subjectKidney Diseases/blooden
dc.subjectKidney Tubules, Proximal/metabolism/*pathologyen
dc.subjectMaleen
dc.subjectUric Acid/*blooden
dc.titleHypouricaemia as a marker of a generalized proximal tubular damage in alcoholic patientsen
heal.abstractAIMS AND METHODS: Hypouricaemia is not frequently encountered in alcoholic patients. Described herein is the case of a 69-year-old alcoholic patient. RESULTS: The patient presented with severe hypouricaemia (serum uric acid 95.2 micro mol/l) due to renal urate wasting associated with a cluster of other metabolic abnormalities in the context of a reversible generalized dysfunction of the proximal tubules that mimicked Fanconi syndrome. CONCLUSIONS: The underlying mechanisms of this rare presentation are discussed.en
heal.accesscampus-
heal.fullTextAvailabilityTRUE-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/12217941-
heal.identifier.secondaryhttp://alcalc.oxfordjournals.org/content/37/5/472.full.pdf-
heal.journalNameAlcohol Alcoholen
heal.journalTypepeer-reviewed-
heal.languageen-
heal.publicationDate2002-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.typejournalArticle-
heal.type.elΆρθρο Περιοδικούel
heal.type.enJournal articleen

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